Collaborations and publications

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Collaborations

Das ursprüngliche Protokoll für die SKIPOGH Studie basiert auf den gleichen Untersuchungen wie die EPOGH Studie (European Project on Genes in Hypertension) und die beiden Projekte sind durch mehrere Forschungsprojekte miteinander verbunden.

Erfreulicherweise kam es in den letzten fünf Jahren noch zu vielen weiteren Kollaborationen. Diese entstanden einerseits durch verschiedene Anfragen, nachdem wir unsere Resultate entweder in wissenschaftlichen Journalen oder an Konferenzen publik gemacht haben, und andrerseits durch das steigende Interesse von Wissenschaftlern an unserer Kohorte, die auf dem gleichen Gebiet arbeiten wie wir. Dieses wissenschaftliche Interesse steigert den Wert der SKIPOGH Studie und zeigt, dass sie auf ihre Art einzigartig ist.

Im nachfolgenden Teil sind die wichtigsten kollaborierenden nationalen und internationalen Forscher aufgeführt:

SKIPOGH participe au projet international EPOGH (European Project on Genes in Hypertension), sur plusieurs axes de recherche.

Plusieurs autres collaborations ont été initiées ces cinq dernières années, qui contribuent à augmenter la valeur de cette étude. L’intérêt grandissant de la communauté scientifique et médicale, les multiples demandes de collaboration qui ont suivi les publications de nos résultats, que ce soit dans des journaux ou lors de conférences, les contacts entretenus avec d’autres chercheurs dans le même domaine, montrent que le projet SKIPOGH est une cohorte populationnelle unique en son genre.

Voici les principales collaborations nationales et internationales à ce jour:

SKIPOGH is a participant of the EPOGH project, and collaborates with this European research at different levels.

Several other collaborations were initiated in the past five years, which greatly increases the value of the SKIPOGH study. The great interest of the scientific community, the multiple requests for collaboration following publications of our results, in journals or conferences, continuous contacts with other researchers in the same domain, show that the SKIPOGH study is a unique and very valuable population-based cohort.

Here are the main other ongoing national and international collaborations:

Collaborations

  • OSAV (www.blv.admin.ch) - Dr. Vincent Dudler. Mesure du Cadmium dans le sang et l’urine
  • H2020 LIFEPATH (www.lifepathproject.eu) - Dr Silvia Stringhini, Dr Cristian Carmelli, Dusan Petrovic for the Swiss part, PI Prof Paolo Vineis, Imperial College, London UK
  • SNF, NCCR-Kidney (www.nccr-kidney.ch ) - Fetuin A, calcium propensity score (Dr Andreas Pasch). Erythropoietin (Prof Roland Wenger). GWAS plateform (Prof Olivier Devuyst, Prof Murielle Bochud)
  • SNF, NCCR-TransCure (www.nccr-transcure.ch) - Dr Cristian Carmeli, Dr Idris Guessous, Prof Murielle Bochud
  • Copeptin - Dr Belen Ponte. Fonds départementaux HUG de Projets & recherche
  • Ouabain - Prof Paolo Manunta, Fondazione Centro San Raffaele del Monte Tabor, Milano, Italy
  • PTH, FGF 23 - Dr Daniel Fuster, Inselspital, Bern. Unrestricted grant from Abbvie
  • Caffeine and other methylxanthines - Prof ass Chin Eap, CHUV
  • 25 Oh VitaminD - Dr. Idris Guessous, UEP HUG
  • MGP - Dr. Cees Vermeer, Vitak R&D Group, University of Maastricht, The Netherlands

Publications

2015

  • Ackermann, D., et al., CYP17A1 Enzyme Activity Is Linked to Ambulatory Blood Pressure in a Family-Based Population Study. Am J Hypertens, 2015. [1]
  • Alwan, H., et al., Heritability of ambulatory and office blood pressure in the Swiss population. J Hypertens, 2015. 33(10): p. 2061-7. [2]
  • Canivell, S., et al., 4B.05: PLASMA COPEPTIN IS ASSOCIATED WITH INSULIN RESISTANCE IN A SWISS POPULATION-BASED STUDY. J Hypertens, 2015. 33 Suppl 1: p. e54. [3]
  • Forni Ogna, V., et al., New anthropometry-based age- and sex-specific reference values for urinary 24-hour creatinine excretion based on the adult Swiss population. BMC Med, 2015. 13: p. 40. [4]
  • Guessous, I., et al., Associations of ambulatory blood pressure with urinary caffeine and caffeine metabolite excretions. Hypertension, 2015. 65(3): p. 691-6. [5]
  • Guessous, I., et al., 1C.06: AMBULATORY PULSE PRESSURE IS NEGATIVELY ASSOCIATED WITH EXCRETIONS OF URINARY CAFFEINE AND ITS METABOLITES. J Hypertens, 2015. 33 Suppl 1: p. e10-1. [6]
  • Moulin, F., et al., 5C.09: HERITABILITY OF RENAL FUNCTION PARAMETERS AND ELECTROLYTE LEVELS IN THE SWISS POPULATION. J Hypertens, 2015. 33 Suppl 1: p. e70. [7]
  • Pivin, E., et al., Inactive Matrix Gla-Protein Is Associated With Arterial Stiffness in an Adult Population-Based Study. Hypertension, 2015. 66(1): p. 85-92. [8]
  • Pivin, E., et al., 1D.03: INACTIVE MATRIX GLA PROTEIN IS ASSOCIATED WITH RENAL RESISTIVE INDEX IN A POPULATION-BASED STUDY. J Hypertens, 2015. 33 Suppl 1: p. e15. [9]
  • Ponte, B., et al., Copeptin is associated with kidney length, renal function, and prevalence of simple cysts in a population-based study. J Am Soc Nephrol, 2015. 26(6): p. 1415-25. [10]
  • Pruijm, M., et al., Associations of Urinary Uromodulin with Clinical Characteristics and Markers of Tubular Function in the General Population. Clin J Am Soc Nephrol, 2015. [11]

Under current revision in Psychoneuroendocrinology (22.12.2015)

  • Petrovic, D., et al. Sociodemographic, behavioral and genetic determinants of allostatic load in a Swiss population-based study

2014

  • Alwan, H., et al., Epidemiology of masked and white-coat hypertension: the family-based SKIPOGH study. PLoS One, 2014. 9(3): p. e92522. [12]
  • Ponte, B., et al., Reference values and factors associated with renal resistive index in a family-based population study. Hypertension, 2014. 63(1): p. 136-42. [13]

2013

  • Ehret, G., et al., Genes for blood pressure: an opportunity to understand hypertension. Eur Heart J, 2013. 34(13): p. 951-61. [14]
  • Pruijm, M., et al., Heritability, determinants and reference values of renal length: a family-based population study. Eur Radiol, 2013. 23(10): p. 2899-905. [15]
  • Trudu, M., et al., Common noncoding UMOD gene variants induce salt-sensitive hypertension and kidney damage by increasing uromodulin expression. Nat Med, 2013. 19(12): p. 1655-60. [16]

2012

  • Ehret, G., et al., A multi-SNP locus-association method reveals a substantial fraction of the missing heritability. Am J Hum Genet, 2012. 91(5): p. 863-71. [17]
  • Guessous, I., et al., Caffeine intake and CYP1A2 variants associated with high caffeine intake protect non-smokers from hypertension. Hum Mol Genet, 2012. 21(14): p. 3283-92. [18]

2011

  • Bochud, M., et al., Public health genomics and the challenges for epidemiology. Eur J Public Health, 2011. 21(1): p. 5-6. [19]
  • Ehret, G., et al., Genetic variants in novel pathways influence blood pressure and cardiovascular disease risk. Nature, 2011. 478(7367): p. 103-9. [20]
  • Guessous, I., et al., Calcium, vitamin D and cardiovascular disease. Kidney Blood Press Res, 2011. 34(6): p. 404-17. [21]

2010

  • Ehret, G., Genome-wide association studies: contribution of genomics to understanding blood pressure and essential hypertension. Curr Hypertens Rep, 2010. 12(1): p. 17-25. [22]
  • Ehret, G., et al., Resistant hypertension. Rev Med Suisse, 2010. 6(262): p. 1721-2, 1724-7. [23]
  • Pruijm, M., et al., A new technique for simultaneous validation of two manual nonmercury auscultatory sphygmomanometers (A&D UM-101 and Accoson Greenlight 300) based on the International protocol. Blood Press Monit, 2010. 15(6): p. 322-5. [24]
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